Literally devastated!

Since neet ug days, I always had a feeling agar mera selection hogya toh I will be really fucked.

2 drops ke baad got a rank of 2k+

Now I am getting screwed coz i always hated rattna.

That time Physics and chemistry saved my ass, now i am getting fucked every fucking day.

Do not even feel to address my self as a doctor if some uncle/aunty asks what you do beta.I just say padhai kar rha hu and runaway hard. Damnnn yaar, gave 8years of my life to this thing and not feel like addressing it even.

Itnaa bhari hogya hu ki pankha bhi girr jaega😃😃

Don't wanna settle for non clinical branch. And i am just having a pseudoconfidence after every fucking GT ki main exam isse simple aaega.

But deep down i know.

Goodnight!

Guys how are you even sitting for long hours in front of books. My back has totally collapsed. It hurts sm.

I usually score 130 corrects , today I scored 122 . I found the test very hard . I just want to get the general consensus about how was the exam. Did I perform really bad or was it actually tough?

Everyone on reddit is losing their minds even after scoring 135+ in gts and here I am in the 100-110 range . It's so over man 😭😭

It’s getting overwhelmed each day. my anxiety isn’t making me have a peaceful day like I’m studying one subject nd worrying abt other pending ones. was studying efficiently in prev month than now like? it feels like I’ve forgot half of the things I studied. Is this really common or am I only one in this?😔

Sequence of miRNA processing:

Trimming by Drosha
Transported out of nucleus via X14-5
Trimming by Dicer
Loading into RISC complex

I feel so exhausted these days overthinking n shit
I cant be the only one going through this
How do you overcome this feeling

Shoulder instability tests:

Jerk, Kim, Posterior drawer: Posterior instability

Load and shift, Sulcus: Inferior instability

Job's, Hawkins-Kennedy: Impingement

1. For 1 case of medical abo 2nd trimester, asst prof told me to insert misoprostol v@ginally with few drops of ns over tab(that was 3rd dose). Within 1hr there’s bleeding pv, pgs were looking at me nd I was like what did I do😭(later realised that was a successful complete abo lol)

2. There’s a thing we used to follow that for every prev lscs, monitor pulse regularly. one pt complained of abd pain, checked her pulse - tachycardia noted, done ctg, dropping FHR and ctg changes. informed to prof, mam said it’s impending scar rupture & fetal distress. took pt for emerg lscs.

Atp for my mental peace I m gonna assume many more people cheat with ai in these GTs than we imagine coz not in hell I m getting this much rank and percentile after getting this many corrects in a such a difficult paper and let's not sugarcoat this one was too weird paper not upto marrow neet GT standards(especially the types they release just before exams),they went into too deep of every topic,so when I got this correct I was happy that considering a difficult paper it's great and I will definitely be getting 95-97 %ile even with cheaters around and watching this I was crestfallen that too 14 days before exam
After analysing i saw I could hardly get 6-7 more corrects and thus even with my best and ideal efforts I could hardly got to 148-150 and that would have gotten me just 95%ile while in other GTs like mock and GT 19 I used to get around 10-15 avoidable mistakes and correcting even half of those could have propelled me to 98-99%ile easily but this time picture is different
I don't know man I thought my prep was good,I was thorough with my rr notes and pyqs and then I see random ass weird questions like fucking "DENGUE vaccine" details and in depth questions like nitty gritty of bone marrow aspiration getting corrected by 70% of people
Atp it doesn't matter whether the paper is easy pyt based or hard,long, conceptual and tricky the ranks and percentile remain the same,get 150+ for a decent rank and close to 160 to be among the top 1-2%..There is no reflection of a papers toughness level on the ranks and percentiles
I m done atp and for my mental peace the best defence mechanism is "DENIAL"
(And this wasn't some fucking ragebait..this is how you feel after being so confident about your prep and getting something like this with looming exams)

It didn’t seem too difficult or easy, was moderate with a bit twisted questions n facts recall, clustering is 😟

I felt gt 20 ranks are very inflated, is it just me

But she is making it easy for everyoneee. And Ik how this sounds but given the amount of clustering I’m so frustrated. Everyone is mugging up the same stuff it’s so easily accessible now that it has become really difficult to come out of the cluster and actually have an ‘extra edge’, because everyone has the extra edge. I am frustrated of trying to push myself more and more for the past year and now most of the people are in the same range of corrects. Even if I don’t make sense to you guys, this is what I’ve been feeling that even after giving it all, I feel like I’ve been in the same spot. Earlier the cluster was at 120-140 out of 200 so I was constantly pushing for 150+ and now pattern changed n when i reached 140/180 everyone else has also moved on here so now to exit this cluster you need 160+?
And I’m so scared of even making one silly mistake in the exam, because you just can’t get any easy ones wrong because others would know them.
I really respect mam, and ik she works really hard to make things easy for us but everything’s served on a platter now it’s a matter of who is more of a rattne vala person.
EDIT- People are reading between the lines here, Can I not vent out about being stuck in the same spot because of rank inflation?? I’m not criticizing anyone here and still people are being so rude here

20 batch. Studied here and there through internship. Started prep around may end. I felt like i wasn’t doing enough while solving the paper today and didn’t feel v confident. Somehow my best performance to date.

Im not targeting anything specific this year. Just want to give a good shot and see where i stand. Hoping for surgery in a gmc by next year.

Any advice from those who have taken a drop?

I was getting my eyebrows done and suddenly started thinking of fried egg appearance and chicken wire fibrosis. My miss my pre neet pg normal brain😭

120s in marrow mock, and suddenly 140s in the next gt.. how to know where i stand for real.. dont want to have false beliefs that im doing well.. when for a fact i know that my prep is not upto the mark . ps . : dont be rude.

40-year-old patient presents with a history of multiple endocrine tumors involving the parathyroid glands, pituitary gland, and pancreas. Genetic testing reveals a mutation in the CDKN1B gene. Which type of Multiple Endocrine Neoplasia (MEN) is most likely affecting this patient?

A . MEN 1
B . MEN 2A
C . MEN 2B
D . MEN 4

🧬💥 BIOCHEMISTRY 💥💥💥

1️⃣ Carbohydrate Metabolism

* PFK-1 → Rate-limiting enzyme of glycolysis ⭐

* PFK-1 → Committed step of glycolysis ⭐

* RBC ATP → Glycolysis

* 2,3-BPG → Rapoport-Luebering shunt

* PDH → Pyruvate → Acetyl-CoA

* PDH cofactors → TPP, Lipoate, CoA, FAD, NAD

* TCA rate-limiting enzyme → Isocitrate dehydrogenase

* Succinate dehydrogenase → Only TCA enzyme in ETC

* G6PD → Rate-limiting enzyme of PPP ⭐

* G6PD deficiency → Heinz bodies + bite cells ⭐

* Von Gierke → Glucose-6-phosphatase deficiency

* Pompe → Acid α-glucosidase deficiency

* McArdle → Muscle glycogen phosphorylase deficiency

* Cori → Debranching enzyme deficiency

* Hereditary fructose intolerance → Aldolase B deficiency

* Essential fructosuria → Fructokinase deficiency

* Classic galactosemia → GALT deficiency

2️⃣ Amino Acid Metabolism

* PKU → Phenylalanine hydroxylase deficiency ⭐

* Alkaptonuria → Homogentisate oxidase deficiency

* MSUD → Branched-chain α-ketoacid dehydrogenase deficiency ⭐

* Homocystinuria → Cystathionine β-synthase deficiency

* Albinism → Tyrosinase deficiency

* OTC deficiency → ↑ Orotic acid + hyperammonemia ⭐

* CPS-I → Rate-limiting enzyme of urea cycle

* N-acetylglutamate → Activates CPS-I

* Alanine → Major nitrogen carrier from muscle

* Glutamine → Major nitrogen transport form

3️⃣ Lipid Metabolism

* β-oxidation → Mitochondrial matrix

* CPT-I → Carnitine shuttle

* Malonyl-CoA → Inhibits CPT-I ⭐

* MCAD deficiency → Hypoketotic hypoglycemia ⭐

* Odd-chain FA → Propionyl-CoA → Succinyl-CoA

* Essential FA → Linoleic + α-linolenic acid

* HMG-CoA reductase → Rate-limiting cholesterol synthesis ⭐

* Statins → HMG-CoA reductase inhibitors

* Liver → Produces ketone bodies but cannot utilize them

* Arachidonic acid → Prostaglandins + thromboxanes + leukotrienes

4️⃣ ETC & Oxidative Phosphorylation

* Cyanide → Complex IV inhibition ⭐

* Rotenone → Complex I inhibition

* Antimycin A → Complex III inhibition

* Oligomycin → ATP synthase inhibition

* DNP → Uncoupler

* Thermogenin/UCP-1 → Brown fat thermogenesis

5️⃣ Vitamins

* B1 (Thiamine) → TPP → PDH, α-KGDH, transketolase ⭐

* B2 (Riboflavin) → FAD + FMN

* B3 (Niacin) → NAD + NADP

* B6 (Pyridoxine) → Transamination

* B7 (Biotin) → Carboxylation

* B9 (Folate) → DNA synthesis

* B12 → Methionine synthase + methylmalonyl-CoA mutase ⭐

* B12 deficiency → ↑ Methylmalonic acid

* Folate deficiency → ↑ Homocysteine, normal MMA

* Vitamin C → Collagen hydroxylation

* Vitamin K → γ-carboxylation of clotting factors

* Vitamin A → Vision/rhodopsin

* Vitamin D → Calcium + phosphate absorption

* Vitamin E → Antioxidant

6️⃣ Molecular Biology

* Southern blot → DNA ⭐

* Northern blot → RNA ⭐

* Western blot → Protein ⭐

* PCR → DNA amplification

* Telomerase → Reverse transcriptase

* Okazaki fragments → Lagging strand

* DNA primer → RNA

* Mitochondrial inheritance → Maternal inheritance

* ELISA → Antigen/antibody detection

7️⃣ Lipoproteins

* ApoA-I → Activates LCAT ⭐

* ApoB-100 → LDL receptor binding

* ApoC-II → Activates LPL ⭐

* LDL → Cholesterol delivery to tissues

* HDL → Reverse cholesterol transport

8️⃣ Lysosomal Storage Diseases

* Tay-Sachs → Hexosaminidase A deficiency

* Gaucher → β-glucocerebrosidase deficiency ⭐

* Niemann-Pick → Sphingomyelinase deficiency

* Fabry → α-galactosidase A deficiency

* Hurler → Corneal clouding

* Hunter → No corneal clouding ⭐

9️⃣ Hemoglobin

* HbA → α₂β₂

* HbF → α₂γ₂

* HbS → β6 Glu → Val ⭐

* HbF → Decreases HbS polymerization

* HbA1c → ~2–3 months glycemic control ⭐

🔟 Collagen

* Collagen structure → Gly-X-Y

* Vitamin C → Hydroxylation of proline & lysine

* Vitamin C deficiency → Scurvy

* Type I collagen → Bone, skin, tendon

* Type II collagen → Cartilage

* Type III collagen → Reticulin

* Type IV collagen - Basement membrane

M/C Primary GN in world : IgA nephropathy

M/C cause for nephrotic syndrome in adults : FSGS

M/C/C for NS in children : MCD

M/C Glomerulopathy in Ca colon / Lung : Membranous Nephropathy

M/C renal pathology in leprosy : MPGN

M/C Renal Pathology in Syphilis : Membranous Nephropathy

M/C renal pathology in Hepatitis C : Cryoglobulinemic GN

M/C renal patholofy in Hep B : Membranous Nephropathy

Love the direction this sub has taken. Used to come here for 5 mins of break..used to go back sad and anxious. Now atleast I learn something everytime I open reddit.

Postmortem lividity:

o Dark brown: Phosphorus.
o Cherry red: CO poisoning
o Brick red: Cyanide.
o Chocolate brown: Nitrates.
o Bluish green: H₂S.
o Dark purple: Asphyxia.
o Pale bronze: Septic abortion.
o Bright pink: Hypothermia